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Also, 44% of patients treated with omeprazole did not respond to clopidogrel, whereas 23% of patients treated with pantoprazole were nonresponders.

Among 1198 patients with acute liver failure enrolled in a US prospective study between 1998 and 2007, 133 were attributed to drug induced liver injury, but none were linked to pantoprazole or other antiulcer medications).

Based on the potential for tumorigenicity shown for Pantoprazole in rodent carcinogenicity studies, a decision should be made whether to discontinue nursing or to discontinue the drug, taking into account the benefit of the drug to the mother.


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High-dose intravenous treatment is the norm; however, high-dose oral therapy may be able to maintain the intragastric pH about 6 as well.

These doses resulted in pharmacodynamic effects on gastric but not esophageal pH.

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Pharmacodynamic parameters were also measured and demonstrated that the change in inhibition of platelet aggregation (induced by 5 µM ADP) was correlated with the change in the exposure to clopidogrel active metabolite.

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